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dc.contributor.authorInan, Sibel
dc.contributor.authorYavas, Guliz
dc.contributor.authorInan, Umit Ubeyt
dc.date.accessioned2021-06-03T06:15:44Z
dc.date.available2021-06-03T06:15:44Z
dc.date.issued2019
dc.identifier.issn2457-4325
dc.identifier.urihttp://dx.doi.org/
dc.identifier.urihttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC6626925/
dc.identifier.urihttp://hdl.handle.net/11655/24245
dc.description.abstractPurpose: To report a case with monocular transient vision loss (TVL) associated with Hyperhomocysteinemia. , Methods: We present a case with persistent TVL attacks and high level of homocysteine. , Results: A 32-year-old male had a history of episodes of recurrent monocular TVL. Extensive ophthalmic, systemic and laboratory studies were unremarkable with the exception of high plasma homocysteine level. He never experienced TVL during the 36-month follow-up after starting folate, B12 and B6 except for one episode in which he had discontinued the treatment for three months. , Conclusion: This case may suggest hyperhomocysteinemia as one of the underlying causes of recurrent attacks of TVL without any known source of emboli.
dc.language.isoen
dc.rightsAttribution 4.0 United States
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.titleHomocysteinemia As A Cause For Amaurosis Fugax In A Patient Without An Apparent Embolic Source
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.relation.journalRomanian Journal Of Ophthalmology
dc.contributor.departmentGöz Hastalıkları
dc.identifier.volume63
dc.identifier.issue2
dc.description.indexPubMed


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Attribution 4.0 United States
Except where otherwise noted, this item's license is described as Attribution 4.0 United States