dc.contributor.author | Inan, Sibel | |
dc.contributor.author | Yavas, Guliz | |
dc.contributor.author | Inan, Umit Ubeyt | |
dc.date.accessioned | 2021-06-03T06:15:44Z | |
dc.date.available | 2021-06-03T06:15:44Z | |
dc.date.issued | 2019 | |
dc.identifier.issn | 2457-4325 | |
dc.identifier.uri | http://dx.doi.org/ | |
dc.identifier.uri | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6626925/ | |
dc.identifier.uri | http://hdl.handle.net/11655/24245 | |
dc.description.abstract | Purpose: To report a case with monocular transient vision loss (TVL) associated with Hyperhomocysteinemia. , Methods: We present a case with persistent TVL attacks and high level of homocysteine. , Results: A 32-year-old male had a history of episodes of recurrent monocular TVL. Extensive ophthalmic, systemic and laboratory studies were unremarkable with the exception of high plasma homocysteine level. He never experienced TVL during the 36-month follow-up after starting folate, B12 and B6 except for one episode in which he had discontinued the treatment for three months. , Conclusion: This case may suggest hyperhomocysteinemia as one of the underlying causes of recurrent attacks of TVL without any known source of emboli. | |
dc.language.iso | en | |
dc.rights | Attribution 4.0 United States | |
dc.rights | info:eu-repo/semantics/openAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by/4.0/ | |
dc.title | Homocysteinemia As A Cause For Amaurosis Fugax In A Patient Without An Apparent Embolic Source | |
dc.type | info:eu-repo/semantics/article | |
dc.type | info:eu-repo/semantics/publishedVersion | |
dc.relation.journal | Romanian Journal Of Ophthalmology | |
dc.contributor.department | Göz Hastalıkları | |
dc.identifier.volume | 63 | |
dc.identifier.issue | 2 | |
dc.description.index | PubMed | |