dc.contributor.author | Mendi, Ayşegül | |
dc.contributor.author | Köse, Sevil | |
dc.contributor.author | Uçkan, Duygu | |
dc.contributor.author | Akca, Gülçin | |
dc.contributor.author | Yilmaz, Derviş | |
dc.contributor.author | Aral, Levent | |
dc.contributor.author | Gültekin, Sibel Elif | |
dc.contributor.author | Eroğlu, Tamer | |
dc.contributor.author | Kiliç, Emine | |
dc.contributor.author | Uçkan, Sina | |
dc.date.accessioned | 2020-01-27T09:30:00Z | |
dc.date.available | 2020-01-27T09:30:00Z | |
dc.date.issued | 2016 | |
dc.identifier.issn | 1678-7757 | |
dc.identifier.uri | https://doi.org/10.1590/1678-775720150145 | |
dc.identifier.uri | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4775012/ | |
dc.identifier.uri | http://hdl.handle.net/11655/21869 | |
dc.description.abstract | An increasing body of evidence suggests that the use of probiotic bacteria is a promising intervention approach for the treatment of inflammatory diseases with a polymicrobial etiology. P. gingivalis has been noted to have a different way of interacting with the innate immune response of the host compared to other pathogenic bacteria, which is a recognized feature that inhibits CXCL8 expression. | tr_TR |
dc.language.iso | en | tr_TR |
dc.publisher | Scientific Electronic Library Online | tr_TR |
dc.relation.isversionof | 10.1590/1678-775720150145 | tr_TR |
dc.rights | info:eu-repo/semantics/openAccess | tr_TR |
dc.subject | CXCL8 chemokine | tr_TR |
dc.subject | Stroma | tr_TR |
dc.subject | Periodontitis | tr_TR |
dc.subject | Probiotics | tr_TR |
dc.subject | Receptor | tr_TR |
dc.subject.lcsh | Tıp | tr_TR |
dc.title | Lactobacillus Rhamnosus Could Inhibit Porphyromonas Gingivalis Derived Cxcl8 Attenuation | tr_TR |
dc.type | info:eu-repo/semantics/article | tr_TR |
dc.type | info:eu-repo/semantics/publishedVersion | |
dc.relation.journal | Journal of Applied Oral Science | tr_TR |
dc.contributor.department | Çocuk Sağlığı ve Hastalıkları | tr_TR |
dc.identifier.volume | 24 | tr_TR |
dc.identifier.issue | 1 | tr_TR |
dc.identifier.startpage | 67 | tr_TR |
dc.identifier.endpage | 75 | tr_TR |
dc.description.index | WoS | tr_TR |
dc.description.index | Scopus | tr_TR |
dc.description.index | PubMed | tr_TR |
dc.funding | Yok | tr_TR |